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Living with death and dying

Description

This unit will explore how knowledge and beliefs about death and encounters with death affect people’s lives. It will also examine the concept of a ‘good death’ from an individual perspective in order to enhance the quality of dying.

Subjects

bad_deaths | health and lifestyle | beliefs | death | dying | good_death | myths | Education | X000

License

Attribution-Noncommercial-Share Alike 2.0 UK: England & Wales Attribution-Noncommercial-Share Alike 2.0 UK: England & Wales http://creativecommons.org/licenses/by-nc-sa/2.0/uk/ http://creativecommons.org/licenses/by-nc-sa/2.0/uk/

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Catalyst for Change: A good death

Description

Section 3 - A Good Death

Subjects

medev | ooer | ukoer | death | terminal illness | preparing for death | end of life | patient experience | good death | end of life pathways | dentistry | A000

License

Attribution-Noncommercial-No Derivative Works 2.0 UK: England & Wales Attribution-Noncommercial-No Derivative Works 2.0 UK: England & Wales http://creativecommons.org/licenses/by-nc-nd/2.0/uk/ http://creativecommons.org/licenses/by-nc-nd/2.0/uk/

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17.582 Civil War (MIT) 17.582 Civil War (MIT)

Description

This course surveys the social science literature on civil war. It studies the origins of civil war, discusses variables affecting duration, and examines termination of conflict. This subject is highly interdisciplinary and covers a wide variety of cases, although with concentration on various Balkan civil wars. This course surveys the social science literature on civil war. It studies the origins of civil war, discusses variables affecting duration, and examines termination of conflict. This subject is highly interdisciplinary and covers a wide variety of cases, although with concentration on various Balkan civil wars.

Subjects

Political science | Political science | social science | social science | civil war | civil war | origins | origins | duration | duration | termination | termination | conflict | conflict | Balkan | Balkan | World Bank | World Bank | Identity | Identity | fear | fear | greed | greed | death | death | intervention | intervention | peace | peace | Columbia | Columbia | Sudan | Sudan | Iraq | Iraq | El Salvador | El Salvador | South Africa | South Africa

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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14.11 Special Topics in Economics: The Challenge of World Poverty (MIT) 14.11 Special Topics in Economics: The Challenge of World Poverty (MIT)

Description

This is a course for those who are interested in the challenge posed by massive and persistent world poverty, have had some economics, and believe that economists might have something useful to say about this question. The questions we will take up include: Is extreme poverty a thing of the past? Why do some countries grow fast and others fall further behind? Does growth help the poor? Does foreign aid help? What can we do about corruption? Should we leave it all to the markets? Should we leave it to the non-governmental organizations (NGOs)? Where is the best place to intervene? How do we deal with the disease burden? How do we improve schools? And many others. This is a course for those who are interested in the challenge posed by massive and persistent world poverty, have had some economics, and believe that economists might have something useful to say about this question. The questions we will take up include: Is extreme poverty a thing of the past? Why do some countries grow fast and others fall further behind? Does growth help the poor? Does foreign aid help? What can we do about corruption? Should we leave it all to the markets? Should we leave it to the non-governmental organizations (NGOs)? Where is the best place to intervene? How do we deal with the disease burden? How do we improve schools? And many others.

Subjects

challenge | challenge | world poverty | world poverty | economics | economics | per capita income | per capita income | health | health | fertility | fertility | mortality | mortality | birth | birth | death | death | microfinance | microfinance | NGOs | NGOs | poor | poor | education | education | colonialism | colonialism | globalization | globalization | corruption | corruption | India | India | Mexico | Mexico | United States | United States | economic growth | economic growth | development | development | credit markets | credit markets | prosperity | prosperity

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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24.02 Moral Problems and the Good Life (MIT) 24.02 Moral Problems and the Good Life (MIT)

Description

This course will focus on issues that arise in contemporary public debate concerning matters of social justice. Topics will likely include: euthanasia, gay marriage, racism and racial profiling, free speech, hunger and global inequality. Students will be exposed to multiple points of view on the topics and will be given guidance in analyzing the moral frameworks informing opposing positions. The goal will be to provide the basis for respectful and informed discussion of matters of common moral concern. This course will focus on issues that arise in contemporary public debate concerning matters of social justice. Topics will likely include: euthanasia, gay marriage, racism and racial profiling, free speech, hunger and global inequality. Students will be exposed to multiple points of view on the topics and will be given guidance in analyzing the moral frameworks informing opposing positions. The goal will be to provide the basis for respectful and informed discussion of matters of common moral concern.

Subjects

pleasure | pleasure | desire | desire | satisfaction | satisfaction | objectivity | objectivity | environmentalism | environmentalism | animal rights | animal rights | immortality | immortality | egoism | egoism | skepticism | skepticism | relativism | relativism | toleration | toleration | utilitarianism | utilitarianism | deontology | deontology | virtue | virtue | moral theory | moral theory | global justice | global justice | equality | equality | social justice | social justice | race | race | gender | gender | poverty | poverty | sex | sex | welfare | welfare | freedom | freedom | famly | famly | vengeance | vengeance | retribution | retribution | reform | reform | punishment | punishment | prison | prison | body | body | Michel Foucault | Michel Foucault | John Stuart Mill | John Stuart Mill | death penalty | death penalty | gay marriage | gay marriage | sexuality | sexuality

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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BE.104J Chemicals in the Environment: Toxicology and Public Health (MIT) BE.104J Chemicals in the Environment: Toxicology and Public Health (MIT)

Description

This course addresses the challenges of defining a relationship between exposure to environmental chemicals and human disease. Course topics include epidemiological approaches to understanding disease causation; biostatistical methods; evaluation of human exposure to chemicals, and their internal distribution, metabolism, reactions with cellular components, and biological effects; and qualitative and quantitative health risk assessment methods used in the U.S. as bases for regulatory decision-making. Throughout the term, students consider case studies of local and national interest. This course addresses the challenges of defining a relationship between exposure to environmental chemicals and human disease. Course topics include epidemiological approaches to understanding disease causation; biostatistical methods; evaluation of human exposure to chemicals, and their internal distribution, metabolism, reactions with cellular components, and biological effects; and qualitative and quantitative health risk assessment methods used in the U.S. as bases for regulatory decision-making. Throughout the term, students consider case studies of local and national interest.

Subjects

biostatistics | biostatistics | risk | risk | risk analysis | risk analysis | risk factor | risk factor | environmental agent | environmental agent | environetics | environetics | cause and effect | cause and effect | pollution | pollution | statistical analysis | statistical analysis | toxic | toxic | genetics | genetics | disease | disease | health | health | EPA | EPA | metabolism | metabolism | endocrine | endocrine | immunity | immunity | uncertainty | uncertainty | mortality | mortality | death rate | death rate | prediction | prediction

License

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17.582 Civil War (MIT) 17.582 Civil War (MIT)

Description

This course surveys the social science literature on civil war. Students will study the origins of civil war, discuss variables that affect the duration of civil war, and examine the termination of conflict. This course is highly interdisciplinary and covers a wide variety of cases. This course surveys the social science literature on civil war. Students will study the origins of civil war, discuss variables that affect the duration of civil war, and examine the termination of conflict. This course is highly interdisciplinary and covers a wide variety of cases.

Subjects

Political science | Political science | social science | social science | civil war | civil war | origins | origins | duration | duration | termination | termination | conflict | conflict | World Bank | World Bank | Identity | Identity | fear | fear | greed | greed | death | death | intervention | intervention | peace | peace

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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17.506 Ethnic Politics II (MIT) 17.506 Ethnic Politics II (MIT)

Description

This course is designed mainly for political science graduate students conducting or considering conducting research on identity politics. While 17.504 Ethnic Politics I is designed as a primarily theoretical course, Ethnic Politics II switches the focus to methods. It aims to familiarize the student with the current conventional approaches as well as major challenges to them. The course discusses definition and measurement issues as well as briefly addressing survey techniques and modeling. This course is designed mainly for political science graduate students conducting or considering conducting research on identity politics. While 17.504 Ethnic Politics I is designed as a primarily theoretical course, Ethnic Politics II switches the focus to methods. It aims to familiarize the student with the current conventional approaches as well as major challenges to them. The course discusses definition and measurement issues as well as briefly addressing survey techniques and modeling.

Subjects

measurement | measurement | ethnic diversity | ethnic diversity | fluidity | fluidity | identity | identity | social identity theory | social identity theory | mechanisms of group comparison | mechanisms of group comparison | memory | memory | death | death | stigma | stigma | prejudice | prejudice | contact hypothesis | contact hypothesis | cascade models | cascade models | identity simulation | identity simulation

License

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Dr. Grayson Speaking to Reporters, Announcing President Wilson's Death Dr. Grayson Speaking to Reporters, Announcing President Wilson's Death

Description

Subjects

death | death | president | president | announcement | announcement | wilson | wilson | reporters | reporters | 1924 | 1924 | carytgrayson | carytgrayson

License

No known copyright restrictions

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7.16 Experimental Molecular Biology: Biotechnology II (MIT) 7.16 Experimental Molecular Biology: Biotechnology II (MIT)

Description

The course applies molecular biology and reverse genetics approaches to the study of apoptosis, or programmed cell death (PCD), in Drosophila cells. RNA interference (RNAi), or double stranded RNA-mediated gene silencing, will be used to inhibit expression of candidate apoptosis-related genes in cultured Drosophila cells. Teams of 2 or 3 students will design and carry out experiments to address questions about the genes involved in the regulation and execution of PCD in this system. Some projects involve the use of DNA damaging agents or other cytotoxic chemicals or drugs to help understand the pathways that control a cell's decision to undergo apoptosis. Instruction and practice in written and oral communication are provided. The course applies molecular biology and reverse genetics approaches to the study of apoptosis, or programmed cell death (PCD), in Drosophila cells. RNA interference (RNAi), or double stranded RNA-mediated gene silencing, will be used to inhibit expression of candidate apoptosis-related genes in cultured Drosophila cells. Teams of 2 or 3 students will design and carry out experiments to address questions about the genes involved in the regulation and execution of PCD in this system. Some projects involve the use of DNA damaging agents or other cytotoxic chemicals or drugs to help understand the pathways that control a cell's decision to undergo apoptosis. Instruction and practice in written and oral communication are provided.

Subjects

RNAi | RNAi | RNA interference | RNA interference | programmed cell death | programmed cell death | Drosophilia | Drosophilia | PCD | PCD | mRNA | mRNA | lab notebook | lab notebook | scientific writing | scientific writing | RT-PCR | RT-PCR | S2 RNA | S2 RNA | S2 | S2 | cell culture | cell culture | genetic transcription | genetic transcription | dsRNA | dsRNA | bioinformatics | bioinformatics

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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7.344 Tumor Suppressor Gene p53: How the Guardian of our Genome Prevents Cancer (MIT) 7.344 Tumor Suppressor Gene p53: How the Guardian of our Genome Prevents Cancer (MIT)

Description

Cancer is a leading cause of death worldwide. Cancer involves uncontrolled cell growth, resistance to cell death, failure to differentiate into a particular cell type, and increased cellular motility. A family of gate-keeper genes, known as tumor suppressor genes, plays important roles in preventing the initiation and progression of cancer. Among these, p53 is the most famous. Because of its essential role in maintaining genomic integrity, p53 is often called the guardian of the genome. During this course, we will study how p53 serves as a pivotal tumor suppressor gene in preventing cancer.This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to disc Cancer is a leading cause of death worldwide. Cancer involves uncontrolled cell growth, resistance to cell death, failure to differentiate into a particular cell type, and increased cellular motility. A family of gate-keeper genes, known as tumor suppressor genes, plays important roles in preventing the initiation and progression of cancer. Among these, p53 is the most famous. Because of its essential role in maintaining genomic integrity, p53 is often called the guardian of the genome. During this course, we will study how p53 serves as a pivotal tumor suppressor gene in preventing cancer.This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to disc

Subjects

tumor suppressor gene | tumor suppressor gene | p53 | p53 | p53 protein | p53 protein | cancer | cancer | cell-growth signals | cell-growth signals | cell cycle regulation | cell cycle regulation | DNA damage | DNA damage | DNA repair | DNA repair | programmed cell death | programmed cell death | apoptosis | apoptosis | genome integrity | genome integrity | oncogenes | oncogenes | p53 mutations | p53 mutations | mouse cancer models | mouse cancer models | Mdm2 | Mdm2 | microRNA | microRNA

License

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7.343 The Radical Consequences of Respiration: Reactive Oxygen Species in Aging and Disease (MIT) 7.343 The Radical Consequences of Respiration: Reactive Oxygen Species in Aging and Disease (MIT)

Description

This course will start with a survey of basic oxygen radical biochemistry followed by a discussion of the mechanisms of action of cellular as well as dietary antioxidants. After considering the normal physiological roles of oxidants, we will examine the effects of elevated ROS and a failure of cellular redox capacity on the rate of organismal and cellular aging as well as on the onset and progression of several major diseases that are often age-related. Topics will include ROS-induced effects on stem cell regeneration, insulin resistance, heart disease, neurodegenerative disorders, and cancer. The role of antioxidants in potential therapeutic strategies for modulating ROS levels will also be discussed. This course is one of many Advanced Undergraduate Seminars offered by the Biology D This course will start with a survey of basic oxygen radical biochemistry followed by a discussion of the mechanisms of action of cellular as well as dietary antioxidants. After considering the normal physiological roles of oxidants, we will examine the effects of elevated ROS and a failure of cellular redox capacity on the rate of organismal and cellular aging as well as on the onset and progression of several major diseases that are often age-related. Topics will include ROS-induced effects on stem cell regeneration, insulin resistance, heart disease, neurodegenerative disorders, and cancer. The role of antioxidants in potential therapeutic strategies for modulating ROS levels will also be discussed. This course is one of many Advanced Undergraduate Seminars offered by the Biology D

Subjects

reactive oxygen species | reactive oxygen species | oxygen | oxygen | ROS | ROS | energy | energy | mitochondria | mitochondria | cell signaling | cell signaling | anti-pathogen | anti-pathogen | oxidative damage | oxidative damage | oncogene | oncogene | antioxidant | antioxidant | insulin resistance | insulin resistance | diabetes | diabetes | stem cell | stem cell | neurodegenerative | neurodegenerative | ischemic | ischemic | ATP | ATP | pathways | pathways | NADPH | NADPH | nox | nox | psd | psd | programmed cell death | programmed cell death | apoptosis | apoptosis | hsc | hsc | hematopoietic | hematopoietic

License

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7.340 Avoiding Genomic Instability: DNA Replication, the Cell Cycle, and Cancer (MIT) 7.340 Avoiding Genomic Instability: DNA Replication, the Cell Cycle, and Cancer (MIT)

Description

In this class we will learn about how the process of DNA replication is regulated throughout the cell cycle and what happens when DNA replication goes awry. How does the cell know when and where to begin replicating its DNA? How does a cell prevent its DNA from being replicated more than once? How does damaged DNA cause the cell to arrest DNA replication until that damage has been repaired? And how is the duplication of the genome coordinated with other essential processes? We will examine both classical and current papers from the scientific literature to provide answers to these questions and to gain insights into how biologists have approached such problems. This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored f In this class we will learn about how the process of DNA replication is regulated throughout the cell cycle and what happens when DNA replication goes awry. How does the cell know when and where to begin replicating its DNA? How does a cell prevent its DNA from being replicated more than once? How does damaged DNA cause the cell to arrest DNA replication until that damage has been repaired? And how is the duplication of the genome coordinated with other essential processes? We will examine both classical and current papers from the scientific literature to provide answers to these questions and to gain insights into how biologists have approached such problems. This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored f

Subjects

cell | cell | genetic material | genetic material | cell death | cell death | tumorigenesis | tumorigenesis | mutations | mutations | genes | genes | DNA replication | DNA replication | cell cycle | cell cycle | damaged DNA | damaged DNA | genome | genome | tumor formation | tumor formation | anti-cancer drugs | anti-cancer drugs | viruses | viruses | cellular controls | cellular controls

License

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7.340 Ubiquitination: The Proteasome and Human Disease (MIT) 7.340 Ubiquitination: The Proteasome and Human Disease (MIT)

Description

This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to discuss and learn about current biological research in a highly interactive setting. This seminar provides a deeper understanding of the post-translational mechanisms evolved by eukaryotic cells to target proteins for degradation. Students learn how proteins are recognized and degraded by specific machinery (the proteasome) through their previous tagging with another small protein, ubiquitin. Additional topics include principles of ubiquitin-proteasome function, its control of the most important cellular pathways, and the implication of this system in different human diseases. Finally, spe This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to discuss and learn about current biological research in a highly interactive setting. This seminar provides a deeper understanding of the post-translational mechanisms evolved by eukaryotic cells to target proteins for degradation. Students learn how proteins are recognized and degraded by specific machinery (the proteasome) through their previous tagging with another small protein, ubiquitin. Additional topics include principles of ubiquitin-proteasome function, its control of the most important cellular pathways, and the implication of this system in different human diseases. Finally, spe

Subjects

ubiquitination | ubiquitination | ubiquitin | ubiquitin | proteasome | proteasome | post-translational mechanisms | post-translational mechanisms | ubiquitin-conjugation system | ubiquitin-conjugation system | neurodegenerative diseases | neurodegenerative diseases | immune response | immune response | cell cycle regulation | cell cycle regulation | apoptosis | apoptosis | signal transduction pathways | signal transduction pathways | tumorigenesis | tumorigenesis | protein degradation | protein degradation | Endoplasmic Reticulum Associated Degradation Pathway | Endoplasmic Reticulum Associated Degradation Pathway | ligases | ligases | translocated proteins | translocated proteins | misfolded proteins | misfolded proteins | trafficking membranes | trafficking membranes | cell cycle control | cell cycle control | programmed cell death | programmed cell death | Huntington's Disease | Huntington's Disease | Von Hippel-Lindau Disease | Von Hippel-Lindau Disease

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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7.342 Cancer Biology: From Basic Research to the Clinic (MIT) 7.342 Cancer Biology: From Basic Research to the Clinic (MIT)

Description

This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to discuss and learn about current biological research in a highly interactive setting. In 1971, President Nixon declared the "War on Cancer," but after three decades the war is still raging. How much progress have we made toward winning the war and what are we doing to improve the fight? Understanding the molecular and cellular events involved in tumor formation, progression, and metastasis is crucial to the development of innovative therapy for cancer patients. Insights into these processes have been gleaned through basic research using biochemical, molecular, and genetic ana This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to discuss and learn about current biological research in a highly interactive setting. In 1971, President Nixon declared the "War on Cancer," but after three decades the war is still raging. How much progress have we made toward winning the war and what are we doing to improve the fight? Understanding the molecular and cellular events involved in tumor formation, progression, and metastasis is crucial to the development of innovative therapy for cancer patients. Insights into these processes have been gleaned through basic research using biochemical, molecular, and genetic ana

Subjects

cancer | cancer | tumor | tumor | metastasis | metastasis | genetic analysis | genetic analysis | cancer biology | cancer biology | model organisms | model organisms | genetic pathways | genetic pathways | uncontrolled growth | uncontrolled growth | tumor suppressor genes | tumor suppressor genes | oncogenes | oncogenes | tumor initiation | tumor initiation | cell cycle | cell cycle | chromosomal aberration | chromosomal aberration | apoptosis | apoptosis | cell death | cell death | signal transduction pathways | signal transduction pathways | proto-oncogene | proto-oncogene | mutation | mutation | DNA mismatch repair | DNA mismatch repair | telomeres | telomeres | mouse models | mouse models | tissue specificity | tissue specificity | malignancy | malignancy | stem cells | stem cells | therapeutic resistance | therapeutic resistance | differentiation | differentiation | caner research | caner research | cancer therapeutics | cancer therapeutics | chemotherapy | chemotherapy

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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7.343 Protein Folding, Misfolding and Human Disease (MIT) 7.343 Protein Folding, Misfolding and Human Disease (MIT)

Description

This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to discuss and learn about current biological research in a highly interactive setting. The instructor for this course, Dr. Kosinski-Collins, is a member of the HHMI Education Group. Maintenance of the complex three-dimensional structure adopted by a protein in the cell is vital for function. Oftentimes, as a consequence of environmental stress, genetic mutation, and/or infection, the folded structure of a protein gets altered and multiple proteins stick and fall out of solution in a process known as aggregation. In many protein aggregation diseases, incorrectly folded proteins self-associate, for This course is one of many Advanced Undergraduate Seminars offered by the Biology Department at MIT. These seminars are tailored for students with an interest in using primary research literature to discuss and learn about current biological research in a highly interactive setting. The instructor for this course, Dr. Kosinski-Collins, is a member of the HHMI Education Group. Maintenance of the complex three-dimensional structure adopted by a protein in the cell is vital for function. Oftentimes, as a consequence of environmental stress, genetic mutation, and/or infection, the folded structure of a protein gets altered and multiple proteins stick and fall out of solution in a process known as aggregation. In many protein aggregation diseases, incorrectly folded proteins self-associate, for

Subjects

protein folding | protein folding | misfolded proteins | misfolded proteins | Mad Cow | Mad Cow | Creutzfedt-Jakob Disease | Creutzfedt-Jakob Disease | Alzheimer's Disease | Alzheimer's Disease | Huntington's Disease | Huntington's Disease | protein aggregation | protein aggregation | self-associate | self-associate | cell death | cell death | dementia | dementia | prions | prions | bovine spongiform encephalopathy | bovine spongiform encephalopathy | kuru | kuru | scrapie | scrapie | protein structure | protein structure | amyloid protein | amyloid protein | amyloidosis | amyloidosis | polyglutamine repeats | polyglutamine repeats | fibrils | fibrils

License

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10.442 Biochemical Engineering (MIT) 10.442 Biochemical Engineering (MIT)

Description

This course focuses on the interaction of chemical engineering, biochemistry, and microbiology. Mathematical representations of microbial systems are featured among lecture topics. Kinetics of growth, death, and metabolism are also covered. Continuous fermentation, agitation, mass transfer, and scale-up in fermentation systems, and enzyme technology round out the subject material. This course focuses on the interaction of chemical engineering, biochemistry, and microbiology. Mathematical representations of microbial systems are featured among lecture topics. Kinetics of growth, death, and metabolism are also covered. Continuous fermentation, agitation, mass transfer, and scale-up in fermentation systems, and enzyme technology round out the subject material.

Subjects

chemical engineering | chemical engineering | biochemistry | biochemistry | microbiology | microbiology | mathematical representations of microbial systems | mathematical representations of microbial systems | kinetics of growth | kinetics of growth | kinetics of death | kinetics of death | kinetics of metabolism | kinetics of metabolism | continuous fermentation | continuous fermentation | agitation | agitation | mass transfer | mass transfer | scale-up in fermentation systems | scale-up in fermentation systems | enzyme technology | enzyme technology

License

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15.072J Queues: Theory and Applications (MIT) 15.072J Queues: Theory and Applications (MIT)

Description

This class deals with the modeling and analysis of queueing systems, with applications in communications, manufacturing, computers, call centers, service industries and transportation. Topics include birth-death processes and simple Markovian queues, networks of queues and product form networks, single and multi-server queues, multi-class queueing networks, fluid models, adversarial queueing networks, heavy-traffic theory and diffusion approximations. The course will cover state of the art results which lead to research opportunities. This class deals with the modeling and analysis of queueing systems, with applications in communications, manufacturing, computers, call centers, service industries and transportation. Topics include birth-death processes and simple Markovian queues, networks of queues and product form networks, single and multi-server queues, multi-class queueing networks, fluid models, adversarial queueing networks, heavy-traffic theory and diffusion approximations. The course will cover state of the art results which lead to research opportunities.

Subjects

modeling | modeling | queueing | queueing | queues | queues | queueing systems | queueing systems | communications | communications | manufacturing | manufacturing | computers | computers | call centers | call centers | service industries | service industries | transportation | transportation | applications | applications | birth-death processes | birth-death processes | markovian queues | markovian queues | networks | networks | single-server | single-server | multi-server | multi-server | multi-class queueing | multi-class queueing | fluid models | fluid models | adversarial queueing | adversarial queueing | heavy-traffic theory | heavy-traffic theory | diffusion approximations | diffusion approximations

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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20.104J Chemicals in the Environment: Toxicology and Public Health (BE.104J) (MIT) 20.104J Chemicals in the Environment: Toxicology and Public Health (BE.104J) (MIT)

Description

This course addresses the challenges of defining a relationship between exposure to environmental chemicals and human disease. Course topics include epidemiological approaches to understanding disease causation; biostatistical methods; evaluation of human exposure to chemicals, and their internal distribution, metabolism, reactions with cellular components, and biological effects; and qualitative and quantitative health risk assessment methods used in the U.S. as bases for regulatory decision-making. Throughout the term, students consider case studies of local and national interest. This course addresses the challenges of defining a relationship between exposure to environmental chemicals and human disease. Course topics include epidemiological approaches to understanding disease causation; biostatistical methods; evaluation of human exposure to chemicals, and their internal distribution, metabolism, reactions with cellular components, and biological effects; and qualitative and quantitative health risk assessment methods used in the U.S. as bases for regulatory decision-making. Throughout the term, students consider case studies of local and national interest.

Subjects

biostatistics | biostatistics | risk | risk | risk analysis | risk analysis | risk factor | risk factor | environmental agent | environmental agent | environetics | environetics | cause and effect | cause and effect | pollution | pollution | statistical analysis | statistical analysis | toxic | toxic | genetics | genetics | disease | disease | health | health | EPA | EPA | metabolism | metabolism | endocrine | endocrine | immunity | immunity | uncertainty | uncertainty | mortality | mortality | death rate | death rate | prediction | prediction | 20.104 | 20.104 | 1.081 | 1.081 | ESD.053 | ESD.053 | BE.104J | BE.104J | BE.104 | BE.104

License

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21H.301 The Ancient World: Greece (MIT) 21H.301 The Ancient World: Greece (MIT)

Description

This course elaborates the history of Ancient Greece from the Bronze Age to the death of Alexander. It covers major social, economic, political, and religious trends. It also includes discussions on Homer, heroism, and the Greek identity; the hoplite revolution and the rise of the city-state; Herodotus, Persia, and the (re)birth of history; Empire, Thucydidean rationalism, and the Peloponnesian War; Platonic constructs; Aristotle, Macedonia, and Hellenism. Emphasis is on use of primary sources in translation. This course elaborates the history of Ancient Greece from the Bronze Age to the death of Alexander. It covers major social, economic, political, and religious trends. It also includes discussions on Homer, heroism, and the Greek identity; the hoplite revolution and the rise of the city-state; Herodotus, Persia, and the (re)birth of history; Empire, Thucydidean rationalism, and the Peloponnesian War; Platonic constructs; Aristotle, Macedonia, and Hellenism. Emphasis is on use of primary sources in translation.

Subjects

History | History | Ancient | Ancient | Greece | Greece | Bronze Age | Bronze Age | death | death | Alexander | Alexander | social | social | economic | economic | political | political | religious | religious | trends | trends | Homer | Homer | heroism | heroism | Greek | Greek | identity | identity | hoplite revolution | hoplite revolution | city-state | city-state | Herodotus | Herodotus | Persia | Persia | Empire | Empire | Thucydidean rationalism | Thucydidean rationalism | Peloponnesian War | Peloponnesian War | Platonic constructs | Platonic constructs | Aristotle | Aristotle | Macedonia | Macedonia | Hellenism | Hellenism | primary sources | primary sources | translation. | translation.

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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24.03 Good Food: The Ethics and Politics of Food Choices (MIT) 24.03 Good Food: The Ethics and Politics of Food Choices (MIT)

Description

This course explores the values (aesthetic, moral, cultural, religious, prudential, political) expressed in the choices of food people eat. It analyzes the decisions individuals make about what to eat, how society should manage food production and consumption collectively, and how reflection on food choices might help resolve conflicts between different values. This course explores the values (aesthetic, moral, cultural, religious, prudential, political) expressed in the choices of food people eat. It analyzes the decisions individuals make about what to eat, how society should manage food production and consumption collectively, and how reflection on food choices might help resolve conflicts between different values.

Subjects

food | food | hunger | hunger | human welfare | human welfare | obesity | obesity | weight | weight | omnivore | omnivore | ethics | ethics | agriculture | agriculture | poverty | poverty | ecology | ecology | locavorism | locavorism | vegan | vegan | vegetarian | vegetarian | speciesism | speciesism | activism | activism | junk food | junk food | gender | gender | animal death | animal death | nutrition | nutrition

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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24.02 Moral Problems and the Good Life (MIT) 24.02 Moral Problems and the Good Life (MIT)

Description

This course will focus on issues that arise in contemporary public debate concerning matters of social justice. Topics will likely include: euthanasia, gay marriage, racism and racial profiling, free speech, hunger and global inequality. Students will be exposed to multiple points of view on the topics and will be given guidance in analyzing the moral frameworks informing opposing positions. The goal will be to provide the basis for respectful and informed discussion of matters of common moral concern. This course will focus on issues that arise in contemporary public debate concerning matters of social justice. Topics will likely include: euthanasia, gay marriage, racism and racial profiling, free speech, hunger and global inequality. Students will be exposed to multiple points of view on the topics and will be given guidance in analyzing the moral frameworks informing opposing positions. The goal will be to provide the basis for respectful and informed discussion of matters of common moral concern.

Subjects

pleasure | pleasure | desire | desire | satisfaction | satisfaction | objectivity | objectivity | environmentalism | environmentalism | animal rights | animal rights | immortality | immortality | egoism | egoism | skepticism | skepticism | relativism | relativism | toleration | toleration | utilitarianism | utilitarianism | deontology | deontology | virtue | virtue | moral theory | moral theory | global justice | global justice | equality | equality | social justice | social justice | race | race | gender | gender | poverty | poverty | sex | sex | welfare | welfare | freedom | freedom | death penalty | death penalty | gay marriage | gay marriage | sexuality | sexuality | pornography | pornography | free speech | free speech | hate speech | hate speech

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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24.200 Ancient Philosophy (MIT) 24.200 Ancient Philosophy (MIT)

Description

This course will acquaint the student with some of the ancient Greek contributions to the Western philosophical and scientific tradition. We will examine a broad range of central philosophical themes concerning: nature, law, justice, knowledge, virtue, happiness, and death. There will be a strong emphasis on analyses of arguments found in the texts. This course will acquaint the student with some of the ancient Greek contributions to the Western philosophical and scientific tradition. We will examine a broad range of central philosophical themes concerning: nature, law, justice, knowledge, virtue, happiness, and death. There will be a strong emphasis on analyses of arguments found in the texts.

Subjects

Philosophy | Philosophy | ancient | ancient | Greek western | Greek western | philosophical | philosophical | scientific | scientific | tradition | tradition | themes | themes | nature | nature | law | law | justice | justice | knowledge | knowledge | virtue | virtue | happiness | happiness | death | death | analysis | analysis | arguments | arguments | text | text

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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RES.STP-001 Science Policy Bootcamp (MIT) RES.STP-001 Science Policy Bootcamp (MIT)

Description

The careers of MIT scientists and engineers are significantly determined by public policy decisions made in Washington by the government. However, their access to information on how this system works is limited. Meanwhile, we increasingly understand that science and technology-based innovation is deeply connected to society's economic growth and its ability to generate societal wellbeing, so the public role of science is growing. This course will examine the public policy behind and the government's role in the science and technology innovation system. Given the challenges to future federal science support, this seminar will aim to equip those planning careers in and around science and technology with a basic background for involvement in science policymaking. This course is offered duri The careers of MIT scientists and engineers are significantly determined by public policy decisions made in Washington by the government. However, their access to information on how this system works is limited. Meanwhile, we increasingly understand that science and technology-based innovation is deeply connected to society's economic growth and its ability to generate societal wellbeing, so the public role of science is growing. This course will examine the public policy behind and the government's role in the science and technology innovation system. Given the challenges to future federal science support, this seminar will aim to equip those planning careers in and around science and technology with a basic background for involvement in science policymaking. This course is offered duri

Subjects

science policy | science policy | globalization | globalization | innovation system | innovation system | "valley of death" | "valley of death" | DARPA | DARPA | energy technology | energy technology | Edison's Invention Factory | Edison's Invention Factory | Bell Labs | Bell Labs | Genetech | Genetech | genome project | genome project | Xerox Parc | Xerox Parc | competitiveness debate | competitiveness debate

License

Content within individual OCW courses is (c) by the individual authors unless otherwise noted. MIT OpenCourseWare materials are licensed by the Massachusetts Institute of Technology under a Creative Commons License (Attribution-NonCommercial-ShareAlike). For further information see http://ocw.mit.edu/terms/index.htm

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The Possibility of Religious-Secular Ethical Engagement Debate 2: Euthanasia

Description

The Possibility of Religious-Secular Ethical Engagement: Euthanasia. Julian Savulescu and Charles Camosy held two public debates in Michaelmas Term 2012 under the series title 'The Possibility of Religious-Secular Ethical Engagement'. Wales; http://creativecommons.org/licenses/by-nc-sa/2.0/uk/

Subjects

uehiro | death | philosophy | euthanasia | ethics | morality | uehiro | death | philosophy | euthanasia | ethics | morality

License

http://creativecommons.org/licenses/by-nc-sa/2.0/uk/

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